1. Basics of Electrocardiography Dr.K.Subramanyam 23-3-2009 2. Outline <ul><li>Review of the conduction system </li></ul><ul><li>ECG leads and recording </li></ul><ul><li>ECG waveforms and intervals </li></ul><ul><li>Normal ECG and its variants </li></ul><ul><li>Interpretation and reporting of an ECG </li></ul> 4. What is an ECG? <ul><li>An ECG is the recording (gram) </li></ul><ul><li>of the electrical activity(electro) generated by the cells of the heart(cardio) that reaches the body surface. </li></ul> 5. Recording ECG William Einthoven 6. Useful in diagnosis of… <ul><li>Cardiac Arrhythmias </li></ul><ul><li>Myocardial ischemia and infarction </li></ul><ul><li>Pericarditis </li></ul><ul><li>Chamber hypertrophy </li></ul><ul><li>Electrolyte disturbances </li></ul><ul><li>Drug effects and toxicity </li></ul> 7. Recording an ECG 8. Basics <ul><li>ECG graphs: </li></ul><ul><ul><li>1 mm squares </li></ul></ul><ul><ul><li>5 mm squares </li></ul></ul><ul><li>Paper Speed: </li></ul><ul><ul><li>25 mm/sec standard </li></ul></ul><ul><li>Voltage Calibration: </li></ul><ul><ul><li>10 mm/mV standard </li></ul></ul> 9. ECG Paper: Dimensions 5 mm 1 mm 0.1 mV 0.04 sec 0.2 sec Speed = rate Voltage ~Mass 10. ECG Leads <ul><li>Leads are electrodes which measure the difference in electrical potential between either: </li></ul>1. Two different points on the body (bipolar leads) 2. One point on the body and a virtual reference point with zero electrical potential, located in the center of the heart (unipolar leads) 11. + - RA RA LL + + - - LA LL LA LEAD II LEAD I LEAD III Remember, the RL is always the ground <ul><li>By changing the arrangement of which arms or legs are positive or negative, three unipolar leads (I, II & III ) can be derived giving three "pictures" of the heart's electrical activity from 3 angles. </li></ul>The Concept of a “Lead” Leads I, II, and III I II III 12. ECG Leads <ul><li>The standard ECG has 12 leads: </li></ul>3 Standard Limb Leads 3 Augmented Limb Leads 6 Precordial Leads The axis of a particular lead represents the viewpoint from which it looks at the heart. 13. ECG LEADS <ul><li>Gold Berger :aV frontal leads </li></ul><ul><li>Wilson & co-workwers :chest leads </li></ul> 14. Standard Limb Leads 15. Precordial Leads 16. Precordial Leads 17. Summary of Leads V 1 -V 6 aVR, aVL, aVF (augmented limb leads) Unipolar - I, II, III (standard limb leads) Bipolar Precordial Leads Limb Leads 18. Limb Leads (Einthoven leads) Einthoven triangle Einthoven Rule I+II+III==0 I+(-II)+III=0 I+III=II 20. Arrangement of Leads on the EKG 21. Anatomic Groups (Septum) 22. Anatomic Groups (Anterior Wall) 23. Anatomic Groups (Lateral Wall) 24. Anatomic Groups (Inferior Wall) 25. Anatomic Groups (Summary) 26. Localising the arterial territory Inferior II, III, aVF Lateral I, AVL, V5-V6 Anterior / Septal V1-V4 27. Standard sites unavailable <ul><li>Patient pathology </li></ul><ul><li>Amputation or burns or bandages should be placed as closely as possible to the standard sites </li></ul> 28. Specific cardiac abnormalities <ul><li>Situs inversus dextrocardia right & left arm electrodes should be reversed </li></ul><ul><li>pre-cordial leads should be recorded from V1R(V2) to V6 </li></ul><ul><li>RVH & RV infarction:V3R & V4R </li></ul> 29. Continuous monitoring <ul><li>Bed side: </li></ul><ul><li>Holter monitoring: </li></ul><ul><li>TMT: Mason Likar system </li></ul> 30. Other practical points <ul><li>Electrodes should be selected for maximum adhesiveness and minimum discomfort,electrical noise,and skin-electrode impedance </li></ul><ul><li>Effective contact between electrode and skin is essential. </li></ul><ul><li>ECG :calibration </li></ul> 31. <ul><li>ECG :paper speed </li></ul><ul><li>Electrical artifacts:external or internal </li></ul><ul><li>external can be minimized by straightening the lead wires </li></ul><ul><li>internal can be due to muscle tremors,shivering ,hiccoughs . </li></ul><ul><li>Supine position </li></ul> 32. Interpretation of an ECG 33. Steps involved <ul><li>Heart Rate </li></ul><ul><li>Rhythm </li></ul><ul><li>Axis </li></ul><ul><li>Wave morphology </li></ul><ul><li>Intervals and segments analysis </li></ul><ul><li>Chamber enlargement </li></ul><ul><li>Specific changes </li></ul> 34. Wave forms 35. Determining the Heart Rate <ul><li>Rule of 300 </li></ul><ul><li>10 Second Rule </li></ul> 36. Rule of 300 <ul><li>Take the number of “big boxes” between neighboring QRS complexes, and divide this into 300. The result will be approximately equal to the rate </li></ul><ul><li>Although fast, this method only works for regular rhythms. </li></ul> 37. The Rule of 300 <ul><li>It may be easiest to memorize the following table: </li></ul>50 6 60 5 75 4 100 3 150 2 300 1 Rate # of big boxes 39. 10 Second Rule <ul><li>As most ECGs record 10 seconds of rhythm per page, one can simply count the number of beats present on the ECG and multiply by 6 to get the number of beats per 60 seconds. </li></ul><ul><li>This method works well for irregular rhythms. </li></ul> 40. QRS axis Dr.K.Subramanyam 9-4-2009 41. Genesis of QRS <ul><li>Initially there is a small vector from left to right through the IVS ,followed by a larger vector from right to left through the free wall of the LV </li></ul> 44. Effect of left oriented lead <ul><li>Small septal vector ,directed away from the positive pole resulting in a small q wave </li></ul><ul><li>Larger vector of the free wall ,directed towards the positive pole resulting in a tall R wave </li></ul> 46. Effect of right oriented lead <ul><li>Small septal vector which is directed towards the positive pole,hence a small r wave </li></ul><ul><li>Large vector of free LV wall which is directed away from the lead and hence a large s wave </li></ul> 47. Transition zone <ul><li>Transition from rS to qR pattern which is usually seen in V3 /V4 </li></ul> 48. Rotation of the heart <ul><li>Around AP axis;here the axis runs through the IVS from the ant to post surface of the heart </li></ul><ul><li>Horizontal position;main body of the LV is oriented upwards and to the left:towards leads I and avL(left axis) </li></ul><ul><li>Vertical position;main body of the LV is oriented to leads II and avF(right and inferior) </li></ul> 51. <ul><li>Around oblique axis;the axis runs through the IVS from apex to base </li></ul><ul><li>Anatomical rotation clock-wise and counter clock wise rotation </li></ul> 52. <ul><li>Counter clock-wise more anterior position of LV </li></ul><ul><li>Results in transition zone shifting to left </li></ul> 54. <ul><li>Clock wise rotation;here th RV assumes a more anterior position so that the IVS lay parallel to the chest wall </li></ul><ul><li>There is a shift of the transition zone to the right </li></ul> 57. The QRS Axis <ul><li>The QRS axis represents the net overall direction of the heart’s electrical activity. </li></ul><ul><li>Abnormalities of axis can hint at: </li></ul><ul><li>Ventricular enlargement </li></ul><ul><li>Conduction blocks (i.e. hemiblocks) </li></ul> 59. The QRS Axis By near-consensus, the normal QRS axis is defined as ranging from -30 ° to +90 ° . -30 ° to -90 ° is referred to as a left axis deviation (LAD) +90 ° to +180 ° is referred to as a right axis deviation (RAD) 61. Determining the Axis <ul><li>The Quadrant Approach </li></ul><ul><li>The Equiphasic Approach </li></ul> 62. Determining the Axis Predominantly Positive Predominantly Negative Equiphasic 63. The Quadrant Approach <ul><li>1. Examine the QRS complex in leads I and aVF to determine if they are predominantly positive or predominantly negative. The combination should place the axis into one of the 4 quadrants below. </li></ul> 65. Example 1 Negative in I, positive in aVF RAD 66. Example 2 Positive in I, negative in aVF Predominantly positive in II Normal Axis (non-pathologic LAD) 68. Example 1 Equiphasic in aVF Predominantly positive in I QRS axis ≈ 0° 69. Example 2 Equiphasic in II Predominantly negative in aVL QRS axis ≈ +150° 70. Using leads I, II, III NEGATIVE NEGATIVE UPRIGHT Pathological Left Axis UPRIGHT UPRIGHT BIPHASIC NEGATIVE NEGATIVE Right Axis NEGATIVE NEGATIVE NEGATIVE Extreme Right Axis NEGATIVE UPRIGHT / BIPHASIC UPRIGHT Physiological Left Axis UPRIGHT UPRIGHT UPRIGHT Normal LEAD 3 LEAD 2 LEAD 1 71. Common causes of LAD <ul><li>May be normal in the elderly and very obese </li></ul><ul><li>Due to high diaphragm during pregnancy, ascites, or ABD tumors </li></ul><ul><li>Inferior wall MI </li></ul><ul><li>Left Anterior Hemiblock </li></ul><ul><li>Left Bundle Branch Block </li></ul><ul><li>WPW Syndrome </li></ul><ul><li>Congenital Lesions </li></ul><ul><li>RV Pacer or RV ectopic rhythms </li></ul><ul><li>Emphysema </li></ul> 72. Common causes of RAD <ul><li>Normal variant </li></ul><ul><li>Right Ventricular Hypertrophy </li></ul><ul><li>Anterior MI </li></ul><ul><li>Right Bundle Branch Block </li></ul><ul><li>Left Posterior Hemiblock </li></ul><ul><li>Left Ventricular ectopic rhythms or pacing </li></ul><ul><li>WPW Syndrome </li></ul> 73. The Normal ECG Dr.K.Subramanyam 30-3-2009 74. Normal Sinus Rhythm <ul><li>Originates in the sinus node </li></ul><ul><li>Rate between 60 and 100 beats per min </li></ul><ul><li>P wave axis of +45 to +65 degrees, ie. Tallest p waves in Lead II </li></ul><ul><li>Monomorphic P waves </li></ul><ul><li>Normal PR interval of 120 to 200 msec </li></ul><ul><li>Normal relationship between P and QRS </li></ul><ul><li>Some sinus arrhythmia is normal </li></ul> 76. Sinus Arrhythmia ECG Characteristics: Presence of sinus P waves Variation of the PP interval which cannot be attributed to either SA nodal block or PACs When the variations in PP interval occur in phase with respiration, this is considered to be a normal variant. When they are unrelated to respiration, they may be caused by the same etiologies leading to sinus bradycardia. 77. Normal P wave <ul><li>Atrial depolarisation </li></ul><ul><li>Duration 80 to 100 msec </li></ul><ul><li>Maximum amplitude 2.5 mm </li></ul><ul><li>Axis +45 to +65 </li></ul><ul><li>Biphasic in lead V1 </li></ul><ul><li>Terminal deflection should not exceed 1 mm in depth and 0.03 sec in duration </li></ul> 78. Normal P wave 79. P’ wave <ul><li>Results in negative wave form in leads II,III and avF </li></ul><ul><li>Axis;-80 to -90 </li></ul><ul><li>Retrograde activation of atria due to impulse arising from or passing through AV node </li></ul> 80. Dome & dart p wave <ul><li>Low left atrial rhythm </li></ul><ul><li>Initial dome-like deflexion and a terminal sharp & spike like deflexion </li></ul> 81. Normal QRS complex <ul><li>Completely negative in lead aVR , maximum positivity in lead II </li></ul><ul><li>rS in right oriented leads and qR in left oriented leads (septal vector) </li></ul><ul><li>Transition zone commonly in V3-V4 </li></ul><ul><li>RV5 > RV6 normally </li></ul><ul><li>Normal duration 50-110 msec, not more than 120 msec </li></ul><ul><li>Physiological q wave not > 0.03 sec </li></ul> 83. <ul><li>ECG showing qR pattern in lead III ,disappears on deep inspiration q wave not significant </li></ul><ul><li>Mech:shift in the QRS axis </li></ul> 84. QRS-T angle <ul><li>The normal t wave axis is similar to the QRS axis </li></ul><ul><li>Normally the QRS-T angle does not exceed 60 deg </li></ul> 85. Amplitude of QRS <ul><li>Depends on the following factors </li></ul><ul><li>1.electrical force generated by the ventricular myocardium </li></ul><ul><li>2.distance of the sensing electrode from the ventricles </li></ul> 86. <ul><li>3.Body build;a thin individual has larger complexes when compared to obese individuals </li></ul><ul><li>4.direction of the frontal QRS axis </li></ul> 87. Normal T wave <ul><li>Same direction as the preceding QRS complex </li></ul><ul><li>Blunt apex with asymmetric limbs </li></ul><ul><li>Height < 5mm in limb leads and <10 mm in precordial leads </li></ul><ul><li>Smooth contours </li></ul><ul><li>May be tall in athletes </li></ul> 89. ST segment <ul><li>Merges smoothly with the proximal limb of the T wave </li></ul><ul><li>No true horizontality </li></ul> 91. Normal u wave <ul><li>Best seen in midprecordial leads </li></ul><ul><li>Height < 10% of preceding T wave </li></ul><ul><li>Isoelectric in lead aVL (useful to measure QTc) </li></ul><ul><li>Rarely exceeds 1 mm in amplitude </li></ul><ul><li>May be tall in athletes (2mm) </li></ul> 93. QT interval <ul><li>Normally corrected for heart rate </li></ul><ul><li>Bazett’s formula </li></ul><ul><li>Normal 350 to 430 msec </li></ul><ul><li>With a normal heart rate (60 to 100), the QT interval should not exceed half of the R-R interval roughly </li></ul> 95. Measurement of QT interval <ul><li>The beginning of the QRS complex is best determined in a lead with an initial q wave </li></ul><ul><li> leads I,II, avL ,V5 or V6 </li></ul><ul><li>QT interval shortens with tachycardia and lengthens with bradycardia </li></ul> 96. Prolonged QTc <ul><li>During sleep </li></ul><ul><li>Hypocalcemia </li></ul><ul><li>Ac myocarditis </li></ul><ul><li>AMI </li></ul><ul><li>Drugs like quinidine,procainamide,tricyclic antidepressants </li></ul><ul><li>Hypothermia </li></ul><ul><li>HOCM </li></ul> 97. <ul><li>Advanced AV block or high degree AV block </li></ul><ul><li>Jervell-Lange –Neilson syndrome </li></ul><ul><li>Romano-ward syndrome </li></ul> 98. Shortened QT <ul><li>Digitalis effect </li></ul><ul><li>Hypercalcemia </li></ul><ul><li>Hyperthermia </li></ul><ul><li>Vagal stimulation </li></ul> 99. Normal standardization <ul><li>1 mV=10 mm </li></ul><ul><li>Will result in perfect right angles at each corner </li></ul> 100. overdamping <ul><li>When the pressure of the stylus is too firm on the paper so that it’s movements are retarded </li></ul><ul><li>The ecg deflexions are inscribed more slowly so that they become fractionally wider </li></ul><ul><li>Results in diminished amplitude of deflexions a small s wave may disappear </li></ul> 101. Underdamping or overshoot <ul><li>When the writing stylus is not pressed firmly enough against the paper </li></ul><ul><li>Results in overshoot of the upswing and downswing of the writing stylus,resulting in sharp spikes at the corners </li></ul><ul><li>Effects:deflexions are inscribed more rapidly resulting in fractionally narrower complexes </li></ul> 102. <ul><li>The ecg deflexions may be increased in amplitude . </li></ul><ul><li>An s wave becomes exaggerated </li></ul> 103. Normal Variants in the ECG 104. <ul><li>Sinus arrhythmia </li></ul><ul><li>Persistent juvenile pattern </li></ul><ul><li>Early repolarisation syndrome </li></ul><ul><li>Non specific T wave changes </li></ul> 105. Persistent juvenile pattern 106. Features of ERPS <ul><li>Vagotonia / athletes’ heart </li></ul><ul><li>Prominent J point </li></ul><ul><li>Concave upwards, minimally elevated ST segments </li></ul><ul><li>Tall symmetrical T waves </li></ul><ul><li>Prominent q waves in left leads </li></ul><ul><li>Tall R waves in left oriented leads </li></ul><ul><li>Prominent u waves </li></ul><ul><li>Rapid precordial transition </li></ul><ul><li>Sinus bradycardia </li></ul><ul><li>E arly R ecognition P revents S treptokinase infusion ! </li></ul> 108. Reporting an ECG 109. 1. Patient Details “ Whose ECG is it ?!” 110. 2. Standardisation and lead placement “Is it properly taken ?” 115. 3. Analysis of Rate, Rhythm and Axis 118. 4. Segment and wave form analysis 121. 5. Chamber enlargements 122. Final Impression “ Does the ECG correlate with the clinical scenario ?” 123. Thank you !